参考文献/References:
[1] Nomiyama O，Yoshie O.A family tree of vertebrate chemokine receptors for a unified nomenclature[J].Dev Comp Immunol,2011,35(7):705-715.
[2] Wang W, Soto H, Oldham ER, et al. Identification of anovel chemokine (CCL28), which binds CCR10 (GPR2)[J].J Biol Chem, 2000, 275: 22313-22323.
[3] 康 玲,彭诗元,秦 源,等.CCL28通过增强MMP-2活力促进滋养细胞浸润功能[J].现代妇产科进展,2017,26(5):337-341.
[4] 周 莹. 趋化因子CCL28在缺氧诱导肝癌细胞侵袭转移中的作用研究[D].复旦大学,2012.
[5] Kunkel Eric J,Kim Chang H,Lazarus Nicole H,et al. CCR10 expression is a common feature of circulating and mucosal epithelial tissue IgA Ab-secreting cells.[J]. Journal of Clinical Investigation,2003,111(7):
[6] Hieshima K, Ohtani H, Shibano M, et al. CCL28 has dual roles in mucosal immunity as a chemokine with broad-spectrum antimicrobial activity[J]. J Immunol, 2003, 170: 1452-1461.
[7] Dimberg J, Hugander A, Wagsater D. Protein expression of the chemokine, CCL28, in human colorectal cancer[J].Int J Oncol, 2006, 28: 315-319.
[8] 孙 颖,姜广水.趋化性细胞因子CCL28在多形性腺瘤中表达变化的研究[J].临床医学工程,2009,16(4):57-59.
[9] 孙 颖,胡以俊,姚丽霞,等.趋化性细胞因子CCL28在人涎腺腺淋巴瘤的表达[J].北京口腔医学,2009,17(4):211-213.
[10] 周 莹,张博恒,殷 欣,等.缺氧微环境中CCL28高表达促进HCCLM3细胞迁移和侵袭[J].复旦学报：医学版,2013,40(3):314-318，334.
[11] 任 丽. 肝癌缺氧微环境CCL28升高的意义及机制研究[D].天津医科大学,2014.
[12] Ren Li,Yu Yang,Wang Li,et al. Hypoxia-induced CCL28 promotes recruitment of regulatory T cells and tumor growth in liver cancer[J]. Oncotarget,2016,7(46):
[13] 林凤娟,杨小利,郭雅洁,等.趋化因子CCL28过表达对人乳腺癌细胞增殖的影响[J].中国癌症杂志,2013,23(3):161-166.
[14] 龚迪和. CCL28、CCR3在乳腺癌中的表达及意义[D].浙江大学,2014.
[15] Yang X L,Liu K Y,Lin F J,et al. CCL28 promotes breast cancer growth and metastasis through MAPK-mediated cellular anti-apoptosis and pro-metastasis.[J]. Oncology Reports,2017,38(3):321-324.
[16] Huang Guichun,Tao Leilei,Shen Sunan,et al. Hypoxia induced CCL28 promotes angiogenesis in lung adenocarcinoma by targeting CCR3 on endothelial cells[J]. Scientific Reports,2016,6:274-276.
[17] 张 楠,李铁军,李鲲鹏,等. 血清肿瘤标志物谱及趋化因子蛋白在肺癌中的表达及预测价值研究[J]. 重庆医学,2017,46(25):3484-3485，3488.
相似文献/References:
[1]李连香,于翠革,陈丽宏,等.miR-99b在新辅助化疗宫颈癌组织中的表达[J].陕西医学杂志,2016,(10):1292.
Li Lianxiang,Yu Cuige,Chen Lihong,et al.[J].,2016,(6):1292.
[2]安改丽,侯磊,李旭,等.miR-145对人乳腺癌MCF-7/ADR细胞耐药逆转的作用机制研究[J].陕西医学杂志,2017,(06):695.
[3]朱紫衣,刘晨霞,江忠勇,等.肿瘤细胞线粒体靶向治疗机制及药物研究进展[J].陕西医学杂志,2021,50(9):1170.[doi:DOI:10.3969/j.issn.1000-7377.2021.09.032]
ZHU Ziyi,LIU Chenxia,JIANG Zhongyong,et al.Research progress of tumor cell mitochondrial targeted therapy mechanism and drugs[J].,2021,50(6):1170.[doi:DOI:10.3969/j.issn.1000-7377.2021.09.032]
[4]张文娟,王琰琰,周静祎,等.微小RNA-181c-5p和Kruppel样因子6在子宫内膜样腺癌组织中的表达及其与患者临床病理特征关系研究[J].陕西医学杂志,2024,(4):552.[doi:DOI:10.3969/j.issn.1000-7377.2024.04.026]
ZHANG Wenjuan,WANG Yanyan,ZHOU Jingyi,et al.Expression of miR-181c-5p and Kruppel-like transcription factor 6 and its relationship with clinicopathological features in endometrioid carcinoma tissues[J].,2024,(6):552.[doi:DOI:10.3969/j.issn.1000-7377.2024.04.026]
[5]陈永华,谢健龙,吴 琴,等.DNA损伤反应和极光激酶家族在肿瘤发生与发展中的作用及关系研究进展[J].陕西医学杂志,2024,(11):1576.[doi:DOI:10.3969/j.issn.1000-7377.2024.11.030]
CHEN Yonghua,XIE Jianlong,WU Qin,et al.Research progress on the role and relationship between DNA damage response and aurora kinase family in tumorigenesis and development[J].,2024,(6):1576.[doi:DOI:10.3969/j.issn.1000-7377.2024.11.030]